During fetal development, if androgens (male sex hormones) are not present, are insufficient in quantity, or are unusable, the brain, body, or both will feminize due to the lack of sufficient male hormones or intolerance to m ... read full comment
During fetal development, if androgens (male sex hormones) are not present, are insufficient in quantity, or are unusable, the brain, body, or both will feminize due to the lack of sufficient male hormones or intolerance to male hormones. If androgens are present, sufficient in quantity, and usable, the brain, body or both will masculinize due to the presence of sufficient tolerated male hormones. In humans, genital-gender/sex arises in the 2nd to 3rd months and brain-gender/sex in the 4th to 5th months of fetal development. The time disparity between the development of neurological and genital sexuality increases the probability of cross-gendering leading to homosexuality and other brain based anomalous gender/sex behaviors.
Genetics and teratology.
Based on an extensive review of the literature on the etiology of homosexuality, biological events predominantly explain the origins of sexual orientation. While some question the ethics of studying genetic issues related to homosexuality, in-utero genetic or neuroendocrinological events play a critical role in cross-gendering. Genetic and in-utero chemically induced sex-altered groups illustrate the paradigm: Turner's Syndrome, Androgen Insensitives, Testicular Feminizing Syndrome, Klinefelter's Syndrome (XnY), progestin-administered females, Adrenogenital Syndrome, "super-males" (XYn), etc. Over 450 species other than humans, in the wild and captivity, exhibit "homosexual" behavior, many with life long bonds. Particularly primates appear to have differing degrees of "homosexual" behavior. The first evidence of the sex-behavior center came from research on mammals. Most research involved surgical interventions and the administration or denial of sex hormones during critical brain differentiation periods. In human female fetuses at risk for masculinizing effects from male hormones, German physicians, using in-utero anti-androgens, have forced female genitalia and brain-gender/sex. Although discussed by Dörner, the use of androgens to force male brain-gender is complex and can potentially result in brain damage, attention deficit disorder, learning disabilities, hyper-activity, and hyper-aggression.
Physiology, anatomy, and psychophysiology.
Numerous studies show male homosexuals in many ways approximate females and significantly differ from male heterosexuals. Comparing male homosexuals to female heterosexuals, similarities include muscle structure, sleep patterns, pelvic openings, carry angle of arm, perception, finger dexterity, noticeable acoustic style, visuo-spatial ability, verbal performance, language lateralization, eating disorders, throw-to-target ability, mental rotation task, E.E.G. readings, response to male pheromones, etc. Compared to heterosexual males, some studies suggest homosexual males appear more prone to left-handedness, stuttering, and reading difficulties, differing brain anatomy related to auditory input, atypical gender and sex behavior, differing lipid levels, more empathy and altruism, larger penile dimension, etc. Among other research showing brain differences between homosexual and heterosexual males, anatomical research at Salk Institute comparing male heterosexual and homosexual brains suggests a differing development of the sex-behavior center in male homosexuals. A more recent UCLA study of the male homosexual brain shows broader differences discriminating male homosexuals from both male and female heterosexuals. Research on transsexuals has also revealed significant differences in brain anatomy and gonadotropin secretions.
Hormonal anomalies.
Direct hormonal evidence shows that male homosexuals and heterosexuals significantly differ from one another. In blind analysis, Margolese and Janiger accurately predicted sexual orientation. Some depressives, unknown to be homosexual, may have a similar difference in sex hormone ratio. Dörner and Gooren injected estrogen into male homosexuals, male heterosexuals, and female heterosexuals with removed ovaries. They found the luteinizing hormone response of male homosexuals almost identical with these females and significantly different from male heterosexuals. Similar results are found in male-to-female transsexuals. Studies, using stringent classification of research subjects, show adult male heterosexuals have significantly higher levels of male hormones than do male homosexuals. In animals with litters along a placental line, female mammals between male mammals cause post-natal sex-stereotypic male behaviors (e.g., sexual mounting and aggression) in these females. Dörner hypothesizes that various in humans prenatal and postnatal hormonal administrations may be used to establish specific brain sex/gender outcomes.
Upsetting a female mammal during gestation can produce cross-gendered offspring. Stress apparently lowers androgens in humans and crowding causes stress resulting in anomalous social behavior and cross-gendered behavior in mammals. A similar phenomenon occurs in post hoc research on some homosexual research subjects. Twice the numbers of male homosexuals were born in war-ravaged Germany during World War II than the six years before or after the war.
Pheromones and Sexual Orientation
The vomeronasal system, adjacent to the olfactory system, mediates sexual interest for both males and females. Findings indicate pheromones play an important role in the identification of sexual objects at a subconscious level. For male homosexuals research has shown a clearly female neurological and social response to pheromones produced by males. In addition, preference for human body odors are influenced by gender and sexual orientation.
n. Genetics and Neuroendocrinology.
Augmenting decades old and contemporary research showing more homosexuals are born as the age of the mother increases, recent research implicates H-Y antigens (resulting from previous male births) increase the probability of adult homosexuality and lack of heterosexual cohabitation by as much as 33 percent. The sibling sex ration, without implicating H-Y antigen, suggests that approximately 10% of the variance in male homosexual behavior can be accounted for sibling sex-ratio. Androgen-insensitives (XY females) and partial-androgen insensitives, illustrate the power of genetics in creating a divergence of genetic gender from both behavioral and anatomic gender. XX females born with male genitalia and behavioral masculinization present a clear case for biologically induced cross gendering. Many chromosomal anomalies (e.g., X deletion ring, ring X or isochrome X, missing X, XXXX, XYY, XX/XY, X0/XY, XXY/XX, XXY/XY, X0/XX, etc.) are contributing factors to sexual orientation, level of intellect, performance of tasks, etc. Identical twins are usually concordant in sexual orientation. Identical twins discordant for sexual orientation can be explained by biological events and reluctance of some to reveal their homosexuality. There is one study, dealing with 61 pairs of twins and three sets of triplets. In one case all the triplets were homosexual. Genetic linkage analysis, using extended families, of the X chromosome suggests some familial male homosexuality is likely genetic and passed down the maternal line. A recent study using only homosexual siblings, found through advertisements in Gay newspapers and magazines, using a limited number of markers without the comparisons in extended families called into question the genetic theory of homosexuality. Studies of the genetic contribution to homosexuality are often marred by poor subject selection, precise definitions of behavior, over generalization, inadequate methodology, etc. Based on many studies of male homosexual twins, using very conservative statistical formulae, heritable factors may account for 44 percent of cases. Research on twins, families, and the adopted implicates genetic or neurohormonal origins for sexual orientation.
The argument against genetic factors because homosexuals seldom marry is fallacious. Male homosexuals, subjected to social pressure to marry, often have offspring. Even if homosexuals did not reproduce, the gene pool would still contain recessive traits, sport genes, and neuro-hormonal influences in-utero leading to cross-gendering.
Surgery and brain compromise.
Evidence from neurosurgery challenges the archetypic myth that persons, based on genital sex determination at birth, will exhibit "gender appropriate" or "heterosexual" behavior in adolescence and adulthood. The first such evidence comes from data on pre-frontal lobotomy patients , some being treated for agitated depression. Following surgery, some patients became "spontaneously" homosexual. In line with Money's comments about sex hormone ratios in some depressives, many of these patients had significant depressive illness. Apparently, when the frontal area of the brain could no longer suppress underlying homosexual drives, these pseudoheterosexuals reverted to their normal homosexual preoccupation. In follow-up, patients reported being able to suppress homosexual urges over a long period. Clinical data shows that as the brain deteriorates with aging, some pseudoheterosexuals are unable to mask their underlying homosexuality.
In some countries, homosexuals have been "treated" surgically to force heterosexual behavior. Psychosurgery of the sex behavior center reduces or eliminates homosexual responding in some homosexual males. Many patients, without psychotherapeutic intervention, become heterosexually active after surgery. Some become asexual and have no sexual interest. Psychosurgery in lesbians results in fewer behavioral changes. After these psychosurgeries, failure to follow-up patients and adverse side-effects are common--including death. A German government commission has called for the elimination of these surgeries. Psychosurgeries to change sexual orientation are not available in the United States.
Some males are accidentally mutilated during circumcision. Because of the ease of a male to female change, they are often sexually reassigned as females. Some of these "females" are masculine in behavior and many are "lesbian" during adulthood.
Research shows changes in sexual orientation can result from viral attack of the human sex-behavior center, head injuries, and tumors. Psychosocial stressors leading to pseudosexuality and neurological insult or injury are apparently the only means of significantly altering the expression of sexual orientation. Excepting neurological changes, no credible sex researcher in the last fifty years has suggested sexual orientation changes after childhood. Most agree biological factors often mediate sexual orientation.55
Psychophysiology.
Autonomic pupillary and penile reflexes, minimally controlled by conscious processes, reliably differentiate between male heterosexuals and homosexuals.56 Male homosexual behavior can be reduced or eliminated using painful electric shock or apomorphine vomiting with or without desensitizing male homosexual to females.57 Ego-dystonic homosexuals "self-referred" or sent by "government social service agencies" often have psychopathology and marked homophobia.58 "Homophobia" is best characterized as a revulsion related to homosexual practices or openly homosexual persons. Many "cured" male homosexuals continue to have powerful homosexual urges without erectile function with sexual partners. Despite protracted aversive intervention, male homosexuals continue to have autonomic pupil responses to male stimuli.59 Aversion therapy and other invasive treatments are punitive60 and pose serious ethical concerns.61
During fetal development, if androgens (male sex hormones) are not present, are insufficient in quantity, or are unusable, the brain, body, or both will feminize due to the lack of sufficient male hormones or intolerance to male hormones. If androgens are present, sufficient in quantity, and usable, the brain, body or both will masculinize due to the presence of sufficient tolerated male hormones. In humans, genital-gender/sex arises in the 2nd to 3rd months and brain-gender/sex in the 4th to 5th months of fetal development. The time disparity between the development of neurological and genital sexuality increases the probability of cross-gendering leading to homosexuality and other brain based anomalous gender/sex behaviors.
Genetics and teratology.
Based on an extensive review of the literature on the etiology of homosexuality, biological events predominantly explain the origins of sexual orientation. While some question the ethics of studying genetic issues related to homosexuality, in-utero genetic or neuroendocrinological events play a critical role in cross-gendering. Genetic and in-utero chemically induced sex-altered groups illustrate the paradigm: Turner's Syndrome, Androgen Insensitives, Testicular Feminizing Syndrome, Klinefelter's Syndrome (XnY), progestin-administered females, Adrenogenital Syndrome, "super-males" (XYn), etc. Over 450 species other than humans, in the wild and captivity, exhibit "homosexual" behavior, many with life long bonds. Particularly primates appear to have differing degrees of "homosexual" behavior. The first evidence of the sex-behavior center came from research on mammals. Most research involved surgical interventions and the administration or denial of sex hormones during critical brain differentiation periods. In human female fetuses at risk for masculinizing effects from male hormones, German physicians, using in-utero anti-androgens, have forced female genitalia and brain-gender/sex. Although discussed by Dörner, the use of androgens to force male brain-gender is complex and can potentially result in brain damage, attention deficit disorder, learning disabilities, hyper-activity, and hyper-aggression.
Physiology, anatomy, and psychophysiology.
Numerous studies show male homosexuals in many ways approximate females and significantly differ from male heterosexuals. Comparing male homosexuals to female heterosexuals, similarities include muscle structure, sleep patterns, pelvic openings, carry angle of arm, perception, finger dexterity, noticeable acoustic style, visuo-spatial ability, verbal performance, language lateralization, eating disorders, throw-to-target ability, mental rotation task, E.E.G. readings, response to male pheromones, etc. Compared to heterosexual males, some studies suggest homosexual males appear more prone to left-handedness, stuttering, and reading difficulties, differing brain anatomy related to auditory input, atypical gender and sex behavior, differing lipid levels, more empathy and altruism, larger penile dimension, etc. Among other research showing brain differences between homosexual and heterosexual males, anatomical research at Salk Institute comparing male heterosexual and homosexual brains suggests a differing development of the sex-behavior center in male homosexuals. A more recent UCLA study of the male homosexual brain shows broader differences discriminating male homosexuals from both male and female heterosexuals. Research on transsexuals has also revealed significant differences in brain anatomy and gonadotropin secretions.
Hormonal anomalies.
Direct hormonal evidence shows that male homosexuals and heterosexuals significantly differ from one another. In blind analysis, Margolese and Janiger accurately predicted sexual orientation. Some depressives, unknown to be homosexual, may have a similar difference in sex hormone ratio. Dörner and Gooren injected estrogen into male homosexuals, male heterosexuals, and female heterosexuals with removed ovaries. They found the luteinizing hormone response of male homosexuals almost identical with these females and significantly different from male heterosexuals. Similar results are found in male-to-female transsexuals. Studies, using stringent classification of research subjects, show adult male heterosexuals have significantly higher levels of male hormones than do male homosexuals. In animals with litters along a placental line, female mammals between male mammals cause post-natal sex-stereotypic male behaviors (e.g., sexual mounting and aggression) in these females. Dörner hypothesizes that various in humans prenatal and postnatal hormonal administrations may be used to establish specific brain sex/gender outcomes.
Upsetting a female mammal during gestation can produce cross-gendered offspring. Stress apparently lowers androgens in humans and crowding causes stress resulting in anomalous social behavior and cross-gendered behavior in mammals. A similar phenomenon occurs in post hoc research on some homosexual research subjects. Twice the numbers of male homosexuals were born in war-ravaged Germany during World War II than the six years before or after the war.
Pheromones and Sexual Orientation
The vomeronasal system, adjacent to the olfactory system, mediates sexual interest for both males and females. Findings indicate pheromones play an important role in the identification of sexual objects at a subconscious level. For male homosexuals research has shown a clearly female neurological and social response to pheromones produced by males. In addition, preference for human body odors are influenced by gender and sexual orientation.
n. Genetics and Neuroendocrinology.
Augmenting decades old and contemporary research showing more homosexuals are born as the age of the mother increases, recent research implicates H-Y antigens (resulting from previous male births) increase the probability of adult homosexuality and lack of heterosexual cohabitation by as much as 33 percent. The sibling sex ration, without implicating H-Y antigen, suggests that approximately 10% of the variance in male homosexual behavior can be accounted for sibling sex-ratio. Androgen-insensitives (XY females) and partial-androgen insensitives, illustrate the power of genetics in creating a divergence of genetic gender from both behavioral and anatomic gender. XX females born with male genitalia and behavioral masculinization present a clear case for biologically induced cross gendering. Many chromosomal anomalies (e.g., X deletion ring, ring X or isochrome X, missing X, XXXX, XYY, XX/XY, X0/XY, XXY/XX, XXY/XY, X0/XX, etc.) are contributing factors to sexual orientation, level of intellect, performance of tasks, etc. Identical twins are usually concordant in sexual orientation. Identical twins discordant for sexual orientation can be explained by biological events and reluctance of some to reveal their homosexuality. There is one study, dealing with 61 pairs of twins and three sets of triplets. In one case all the triplets were homosexual. Genetic linkage analysis, using extended families, of the X chromosome suggests some familial male homosexuality is likely genetic and passed down the maternal line. A recent study using only homosexual siblings, found through advertisements in Gay newspapers and magazines, using a limited number of markers without the comparisons in extended families called into question the genetic theory of homosexuality. Studies of the genetic contribution to homosexuality are often marred by poor subject selection, precise definitions of behavior, over generalization, inadequate methodology, etc. Based on many studies of male homosexual twins, using very conservative statistical formulae, heritable factors may account for 44 percent of cases. Research on twins, families, and the adopted implicates genetic or neurohormonal origins for sexual orientation.
The argument against genetic factors because homosexuals seldom marry is fallacious. Male homosexuals, subjected to social pressure to marry, often have offspring. Even if homosexuals did not reproduce, the gene pool would still contain recessive traits, sport genes, and neuro-hormonal influences in-utero leading to cross-gendering.
Surgery and brain compromise.
Evidence from neurosurgery challenges the archetypic myth that persons, based on genital sex determination at birth, will exhibit "gender appropriate" or "heterosexual" behavior in adolescence and adulthood. The first such evidence comes from data on pre-frontal lobotomy patients , some being treated for agitated depression. Following surgery, some patients became "spontaneously" homosexual. In line with Money's comments about sex hormone ratios in some depressives, many of these patients had significant depressive illness. Apparently, when the frontal area of the brain could no longer suppress underlying homosexual drives, these pseudoheterosexuals reverted to their normal homosexual preoccupation. In follow-up, patients reported being able to suppress homosexual urges over a long period. Clinical data shows that as the brain deteriorates with aging, some pseudoheterosexuals are unable to mask their underlying homosexuality.
In some countries, homosexuals have been "treated" surgically to force heterosexual behavior. Psychosurgery of the sex behavior center reduces or eliminates homosexual responding in some homosexual males. Many patients, without psychotherapeutic intervention, become heterosexually active after surgery. Some become asexual and have no sexual interest. Psychosurgery in lesbians results in fewer behavioral changes. After these psychosurgeries, failure to follow-up patients and adverse side-effects are common--including death. A German government commission has called for the elimination of these surgeries. Psychosurgeries to change sexual orientation are not available in the United States.
Some males are accidentally mutilated during circumcision. Because of the ease of a male to female change, they are often sexually reassigned as females. Some of these "females" are masculine in behavior and many are "lesbian" during adulthood.
Research shows changes in sexual orientation can result from viral attack of the human sex-behavior center, head injuries, and tumors. Psychosocial stressors leading to pseudosexuality and neurological insult or injury are apparently the only means of significantly altering the expression of sexual orientation. Excepting neurological changes, no credible sex researcher in the last fifty years has suggested sexual orientation changes after childhood. Most agree biological factors often mediate sexual orientation.55
Psychophysiology.
Autonomic pupillary and penile reflexes, minimally controlled by conscious processes, reliably differentiate between male heterosexuals and homosexuals.56 Male homosexual behavior can be reduced or eliminated using painful electric shock or apomorphine vomiting with or without desensitizing male homosexual to females.57 Ego-dystonic homosexuals "self-referred" or sent by "government social service agencies" often have psychopathology and marked homophobia.58 "Homophobia" is best characterized as a revulsion related to homosexual practices or openly homosexual persons. Many "cured" male homosexuals continue to have powerful homosexual urges without erectile function with sexual partners. Despite protracted aversive intervention, male homosexuals continue to have autonomic pupil responses to male stimuli.59 Aversion therapy and other invasive treatments are punitive60 and pose serious ethical concerns.61
During fetal development, if androgens (male sex hormones) are not present, are insufficient in quantity, or are unusable, the brain, body, or both will feminize due to the lack of sufficient male hormones or intolerance to male hormones. If androgens are present, sufficient in quantity, and usable, the brain, body or both will masculinize due to the presence of sufficient tolerated male hormones. In humans, genital-gender/sex arises in the 2nd to 3rd months and brain-gender/sex in the 4th to 5th months of fetal development. The time disparity between the development of neurological and genital sexuality increases the probability of cross-gendering leading to homosexuality and other brain based anomalous gender/sex behaviors.
Genetics and teratology.
Based on an extensive review of the literature on the etiology of homosexuality, biological events predominantly explain the origins of sexual orientation. While some question the ethics of studying genetic issues related to homosexuality, in-utero genetic or neuroendocrinological events play a critical role in cross-gendering. Genetic and in-utero chemically induced sex-altered groups illustrate the paradigm: Turner's Syndrome, Androgen Insensitives, Testicular Feminizing Syndrome, Klinefelter's Syndrome (XnY), progestin-administered females, Adrenogenital Syndrome, "super-males" (XYn), etc. Over 450 species other than humans, in the wild and captivity, exhibit "homosexual" behavior, many with life long bonds. Particularly primates appear to have differing degrees of "homosexual" behavior. The first evidence of the sex-behavior center came from research on mammals. Most research involved surgical interventions and the administration or denial of sex hormones during critical brain differentiation periods. In human female fetuses at risk for masculinizing effects from male hormones, German physicians, using in-utero anti-androgens, have forced female genitalia and brain-gender/sex. Although discussed by Dörner, the use of androgens to force male brain-gender is complex and can potentially result in brain damage, attention deficit disorder, learning disabilities, hyper-activity, and hyper-aggression.
Physiology, anatomy, and psychophysiology.
Numerous studies show male homosexuals in many ways approximate females and significantly differ from male heterosexuals. Comparing male homosexuals to female heterosexuals, similarities include muscle structure, sleep patterns, pelvic openings, carry angle of arm, perception, finger dexterity, noticeable acoustic style, visuo-spatial ability, verbal performance, language lateralization, eating disorders, throw-to-target ability, mental rotation task, E.E.G. readings, response to male pheromones, etc. Compared to heterosexual males, some studies suggest homosexual males appear more prone to left-handedness, stuttering, and reading difficulties, differing brain anatomy related to auditory input, atypical gender and sex behavior, differing lipid levels, more empathy and altruism, larger penile dimension, etc. Among other research showing brain differences between homosexual and heterosexual males, anatomical research at Salk Institute comparing male heterosexual and homosexual brains suggests a differing development of the sex-behavior center in male homosexuals. A more recent UCLA study of the male homosexual brain shows broader differences discriminating male homosexuals from both male and female heterosexuals. Research on transsexuals has also revealed significant differences in brain anatomy and gonadotropin secretions.
Hormonal anomalies.
Direct hormonal evidence shows that male homosexuals and heterosexuals significantly differ from one another. In blind analysis, Margolese and Janiger accurately predicted sexual orientation. Some depressives, unknown to be homosexual, may have a similar difference in sex hormone ratio. Dörner and Gooren injected estrogen into male homosexuals, male heterosexuals, and female heterosexuals with removed ovaries. They found the luteinizing hormone response of male homosexuals almost identical with these females and significantly different from male heterosexuals. Similar results are found in male-to-female transsexuals. Studies, using stringent classification of research subjects, show adult male heterosexuals have significantly higher levels of male hormones than do male homosexuals. In animals with litters along a placental line, female mammals between male mammals cause post-natal sex-stereotypic male behaviors (e.g., sexual mounting and aggression) in these females. Dörner hypothesizes that various in humans prenatal and postnatal hormonal administrations may be used to establish specific brain sex/gender outcomes.
Upsetting a female mammal during gestation can produce cross-gendered offspring. Stress apparently lowers androgens in humans and crowding causes stress resulting in anomalous social behavior and cross-gendered behavior in mammals. A similar phenomenon occurs in post hoc research on some homosexual research subjects. Twice the numbers of male homosexuals were born in war-ravaged Germany during World War II than the six years before or after the war.
Pheromones and Sexual Orientation
The vomeronasal system, adjacent to the olfactory system, mediates sexual interest for both males and females. Findings indicate pheromones play an important role in the identification of sexual objects at a subconscious level. For male homosexuals research has shown a clearly female neurological and social response to pheromones produced by males. In addition, preference for human body odors are influenced by gender and sexual orientation.
n. Genetics and Neuroendocrinology.
Augmenting decades old and contemporary research showing more homosexuals are born as the age of the mother increases, recent research implicates H-Y antigens (resulting from previous male births) increase the probability of adult homosexuality and lack of heterosexual cohabitation by as much as 33 percent. The sibling sex ration, without implicating H-Y antigen, suggests that approximately 10% of the variance in male homosexual behavior can be accounted for sibling sex-ratio. Androgen-insensitives (XY females) and partial-androgen insensitives, illustrate the power of genetics in creating a divergence of genetic gender from both behavioral and anatomic gender. XX females born with male genitalia and behavioral masculinization present a clear case for biologically induced cross gendering. Many chromosomal anomalies (e.g., X deletion ring, ring X or isochrome X, missing X, XXXX, XYY, XX/XY, X0/XY, XXY/XX, XXY/XY, X0/XX, etc.) are contributing factors to sexual orientation, level of intellect, performance of tasks, etc. Identical twins are usually concordant in sexual orientation. Identical twins discordant for sexual orientation can be explained by biological events and reluctance of some to reveal their homosexuality. There is one study, dealing with 61 pairs of twins and three sets of triplets. In one case all the triplets were homosexual. Genetic linkage analysis, using extended families, of the X chromosome suggests some familial male homosexuality is likely genetic and passed down the maternal line. A recent study using only homosexual siblings, found through advertisements in Gay newspapers and magazines, using a limited number of markers without the comparisons in extended families called into question the genetic theory of homosexuality. Studies of the genetic contribution to homosexuality are often marred by poor subject selection, precise definitions of behavior, over generalization, inadequate methodology, etc. Based on many studies of male homosexual twins, using very conservative statistical formulae, heritable factors may account for 44 percent of cases. Research on twins, families, and the adopted implicates genetic or neurohormonal origins for sexual orientation.
The argument against genetic factors because homosexuals seldom marry is fallacious. Male homosexuals, subjected to social pressure to marry, often have offspring. Even if homosexuals did not reproduce, the gene pool would still contain recessive traits, sport genes, and neuro-hormonal influences in-utero leading to cross-gendering.
Surgery and brain compromise.
Evidence from neurosurgery challenges the archetypic myth that persons, based on genital sex determination at birth, will exhibit "gender appropriate" or "heterosexual" behavior in adolescence and adulthood. The first such evidence comes from data on pre-frontal lobotomy patients , some being treated for agitated depression. Following surgery, some patients became "spontaneously" homosexual. In line with Money's comments about sex hormone ratios in some depressives, many of these patients had significant depressive illness. Apparently, when the frontal area of the brain could no longer suppress underlying homosexual drives, these pseudoheterosexuals reverted to their normal homosexual preoccupation. In follow-up, patients reported being able to suppress homosexual urges over a long period. Clinical data shows that as the brain deteriorates with aging, some pseudoheterosexuals are unable to mask their underlying homosexuality.
In some countries, homosexuals have been "treated" surgically to force heterosexual behavior. Psychosurgery of the sex behavior center reduces or eliminates homosexual responding in some homosexual males. Many patients, without psychotherapeutic intervention, become heterosexually active after surgery. Some become asexual and have no sexual interest. Psychosurgery in lesbians results in fewer behavioral changes. After these psychosurgeries, failure to follow-up patients and adverse side-effects are common--including death. A German government commission has called for the elimination of these surgeries. Psychosurgeries to change sexual orientation are not available in the United States.
Some males are accidentally mutilated during circumcision. Because of the ease of a male to female change, they are often sexually reassigned as females. Some of these "females" are masculine in behavior and many are "lesbian" during adulthood.
Research shows changes in sexual orientation can result from viral attack of the human sex-behavior center, head injuries, and tumors. Psychosocial stressors leading to pseudosexuality and neurological insult or injury are apparently the only means of significantly altering the expression of sexual orientation. Excepting neurological changes, no credible sex researcher in the last fifty years has suggested sexual orientation changes after childhood. Most agree biological factors often mediate sexual orientation.55
Psychophysiology.
Autonomic pupillary and penile reflexes, minimally controlled by conscious processes, reliably differentiate between male heterosexuals and homosexuals.56 Male homosexual behavior can be reduced or eliminated using painful electric shock or apomorphine vomiting with or without desensitizing male homosexual to females.57 Ego-dystonic homosexuals "self-referred" or sent by "government social service agencies" often have psychopathology and marked homophobia.58 "Homophobia" is best characterized as a revulsion related to homosexual practices or openly homosexual persons. Many "cured" male homosexuals continue to have powerful homosexual urges without erectile function with sexual partners. Despite protracted aversive intervention, male homosexuals continue to have autonomic pupil responses to male stimuli.59 Aversion therapy and other invasive treatments are punitive60 and pose serious ethical concerns.61
Mozato 9 years ago
Who are you educating here? Keep your chew and pour to yourself.
Who are you educating here? Keep your chew and pour to yourself.
Mike togbe afede 9 years ago
I went to a party last week, and all I saw were Ghanaian women dancing and kissing each other. Ghanaians are sick apes. They need training from TOGO.
I went to a party last week, and all I saw were Ghanaian women dancing and kissing each other. Ghanaians are sick apes. They need training from TOGO.
WISE COUNSEL 9 years ago
Two times failed its not equal to a win .go back to the maths.the news is important not the writer. Kill the word do not kill the massenger.see you later
Two times failed its not equal to a win .go back to the maths.the news is important not the writer. Kill the word do not kill the massenger.see you later
Kwame Ninsin 9 years ago
You're dumber than a rock. Margaret Jackson is a well-known longstanding activist on Ghanaweb long before Koku Anyidoho was plucked from obscurity in London by Mills.
Just because you make assertions with bravado doesn't mea ... read full comment
You're dumber than a rock. Margaret Jackson is a well-known longstanding activist on Ghanaweb long before Koku Anyidoho was plucked from obscurity in London by Mills.
Just because you make assertions with bravado doesn't mean you're right. It makes you look very dumb
NICHOLAS 9 years ago
If you are a man with strong balls then tell us your real name. Rockson Adofo is certainly not your name!!
If you are a man with strong balls then tell us your real name. Rockson Adofo is certainly not your name!!
Cosmos 9 years ago
Before all the noise Rockson is making about Kumawu was caption under the fake name " JOHN FOSU"
Before all the noise Rockson is making about Kumawu was caption under the fake name " JOHN FOSU"
Akwele 9 years ago
More to the point. Are you back again?
More to the point. Are you back again?
Abeeku Mensah 9 years ago
Look who is talking trash? People in glass houses do not throw stones, Adofo Rockson and that's a wisdom for the day. Sir John and some of the so called "bad boys" or shameless mean-spirited tribal zealots in the NPP or NDC ... read full comment
Look who is talking trash? People in glass houses do not throw stones, Adofo Rockson and that's a wisdom for the day. Sir John and some of the so called "bad boys" or shameless mean-spirited tribal zealots in the NPP or NDC are known and recognizable by face in Ghana. I'm also sure Koku Anyidohu can be picked out of a lineup in Ghana any day. Can you say the same of yourself except hide in your crawl space only to come out and throw stones when convenient? Show yourself in Ghana and walk the talk. Until then your rehearsal for a Nana Akufo-Addo appointment is delusional.
During fetal development, if androgens (male sex hormones) are not present, are insufficient in quantity, or are unusable, the brain, body, or both will feminize due to the lack of sufficient male hormones or intolerance to m ...
read full comment
Who are you educating here? Keep your chew and pour to yourself.
I went to a party last week, and all I saw were Ghanaian women dancing and kissing each other. Ghanaians are sick apes. They need training from TOGO.
Two times failed its not equal to a win .go back to the maths.the news is important not the writer. Kill the word do not kill the massenger.see you later
You're dumber than a rock. Margaret Jackson is a well-known longstanding activist on Ghanaweb long before Koku Anyidoho was plucked from obscurity in London by Mills.
Just because you make assertions with bravado doesn't mea ...
read full comment
If you are a man with strong balls then tell us your real name. Rockson Adofo is certainly not your name!!
Before all the noise Rockson is making about Kumawu was caption under the fake name " JOHN FOSU"
More to the point. Are you back again?
Look who is talking trash? People in glass houses do not throw stones, Adofo Rockson and that's a wisdom for the day. Sir John and some of the so called "bad boys" or shameless mean-spirited tribal zealots in the NPP or NDC ...
read full comment